Thursday, December 23

Facing The Future

Republicans are flipping out after yesterday's court verdict and the likelihood that Gregoire didn't need it to win. That said, the election still hasn't been certified, and it will almost certainly face more court challenges from the Rossi camp. You know, the people who trust the voters more than the lawyers, or so they said when they were on top.

At any rate, whoever wins the governorship, they're going to have to deal with this. I'm not sure how, but my name wasn't on the ballot in November, and aren't we all lucky for that:

...A national report released yesterday, called "Out of Reach 2004," states that a full-time worker in King County must earn $16.04 an hour -- more than twice the state minimum wage of $7.16 -- in order to rent a modest two-bedroom apartment.

This "housing wage" is the amount a person working full-time must earn to afford the fair-market rent on a two-bedroom unit without paying more than 30 percent of his or her income in rent.

Minimum-wage workers in the county must work 90 hours per week to afford a two-bedroom unit, the report says, while statewide, minimum-wage earners are shut out of fair-market rent for even a one-bedroom unit. ...

Rising housing costs, particularly in the greater Puget Sound area, combined with low-paying jobs have forced many workers to pay half or more of their income to afford a roof over their heads, local and national housing experts said. Ideally, workers should not have to pay more than 30 percent of their income for rent, leaving money left over for health care, food, child care, transportation, savings and other living or emergency expenses, experts say.

Yet in Washington, the report says, a minimum-wage worker must work 80 hours per week to afford a two-bedroom unit at the fair-market rent. ...

...Bill Apgar, senior scholar at the Joint Center for Housing Studies at Harvard University, said while there "remains a broadly held perception that folks who can't afford rent are somehow deadbeats," the report is a stark reminder "that you can work full time and have a reasonable income and not even come close to an adequate home." ...



A Daily Kos diarist dealt with the same issue the other day. Though they were talking specifically about the cost of living in other states, the story is a familiar one all across the country:

...I am mainly looking at things over the last three decades (I was born in 1973). As an example, just before I was born, my father had gotten his masters degree in electrical engineering from Penn State University (a good school). He then moved to California and secured a job with a large firm out there with a starting salary of $18,000. In that same year he bought a house in Lomita, California for $30,000 and a used Porsche 911 Carrera (1 year old) for $6,000. In other words, for his one year worth of labor (ignoring taxes for the moment) he was able to purchase 60% of a house in a nice neighborhood in California or 3 Porsche 911's. Fast forward to today, and I have a friend that graduated with a master's degree in engineering from Princeton University (an even better school) in 2002. He was getting offers for starting salaries for around $60,000 or 3.3 x what my dad started out at. Here is a link showing starting salaries for electrical engineers in Los Angeles, CA. from monster.com...

As you can see, it is right around the $60,000 mark. However, based on today's prices, the SAME house in Lomita, CA. is selling for over $400,000 (and the neighborhood has declined) and a used Porsche 911 sells for around $60,000 (the lowest price I could find on autotrader.com for a 3 year old 911 was 56,000). So while salaries for the same professional job have risen by 3.3x, prices for comparable homes and cars in the same area have risen by factors of 13x and 10x, respectively. This means that for the same year of labor that my father had to give up, my friend could only purchase 15% of the same house (in a declining neighborhood) or only 1 Porsche 911 (as opposed to 3). Similar ratios apply to non-luxury cars as well. You could buy a new car from GM or Ford in 1973 for a couple thousand dollars and today you are lucky if you can find a new GM or Ford for under $20,000. The biggest family expenditures are housing, transportation, education and healthcare and all of the big four have been compounding at higher rates than the general level of wages for decades. In the case of education and healthcare, the rate of increase has actually been ACCELERATING over the last four or five years with no end in sight.

Another way to look at wages is to look at the minimum wage. The national minimum wage in 1973 was $1.90 and today in 2004 it is $5.15. That is an increase of 2.7 x over the last 31 years. ...

But the national average median sales price for an existing single family home in 1973 was $28,900 and the national average median price as of the third quarter of 2002 was $218,000. So while minimum wage levels have increased by a factor of 2.7x, the average median home price has grown by a factor of 7.5 x OVER THE SAME TIME PERIOD. ...

Here is another story. My grandparents bought their home in 1955 in a nice neighborhood in a Philadelphia suburb for $14,000. The home was brand new construction. My grandfather was a bus driver his entire life and retired with a pension. My grandmother did not work full time, but did work as a cashier a few hours a week for some extra spending money. They did not live large, but they had enough (on a bus drivers salary) to pay off their home comfortably within ten years (not thirty), take month long summer vacations most years, raise two kids and put their kids through college AND STILL SAVE REGULARLY. Fast forward to today, and they have recently sold their home for $290,000 despite the fact that: (1) the house is now 50 years old, (2) the neighborhood has declined, and (3) they have not made any additions or major renovations since they bought it other than to enclose a small back porch. Based on that price, a bus driver today could not even afford to LOOK at the house, much less pay it off comfortably in ten years!! So my grandfather was a bus driver and it bought him a certain quality of life that today's bus drivers cannot have. I am not begrudging him, just pointing out the reality of the situation.

So we have wage factors for the average family growing at rate X and cost factors (for the major expenditures) growing at multiples of X. This has been happening over decades and it is analogous to putting a frog in a pot of water and slowly turning up the heat. If you picture a V turned on its side, that is what the chart looks like as there is an ever widening gap between wages and costs. The American family has responded to this growing gap by eliminating savings, taking on more debt to support consumption and swelling the ranks of two income households. The housing bubble has pushed the day of reckoning forward as home equity has been tapped to support consumption / pay bills over the last few years like never before.

Where is all this leading?

Well, if the now established trend lines continue, with costs compounding a couple of percentage points faster than wages for another 20 or 30 years, then something has to give. The average family has gone from living comfortably on one blue collar wage to just getting by with two non-blue collar wages. There is no more room to run. ...



We have troubling issues to face as a nation, but with the current administration in power, the states are going to be very much on their own. I hope our next governor is up to the task of working on these issues with us, because no matter who it is, there will be no free pass on coming up with creative solutions to these problems.

Inauguration day is January 12th. January 13th, we start holding feet to the fire, metaphorically speaking. We'll give the poor blighter a few years before we come with the pitchforks ;)

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6:45 AM  
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Anonymous Anonymous said...

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as follows:

CASINO CLASSIC


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Anonymous Anonymous said...

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Hi

Enjoyed the blog - nice to see someone else has activated their anonymous feature ;-)

I'm interested in investing in the real estate market in the US and am looking for some really cheap repossession properties in Miami, Florida, California, New York, Washington, Dallas, Texas, Chicago and Los Angeles. I've found a couple of really good sites - Bargain network which has some really cheap homes and Texas Ranches for sale - ranches in Texas plus both sites have beachfront villas and cars that have been repossessed and are really cheap!! Just wondering if there are any more sites like this.

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4:25 PM  
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Anonymous Anonymous said...

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Hi matey

I'm sure it was this blog I was reading a while back where someone was looking for an SEO tool for driving more traffic and get more hits for their website.

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Anonymous Anonymous said...

asbestos


Mesothelioma
is a form of
cancer that is almost always caused by previous exposure to asbestos.
In this disease, malignant cells develop in the mesothelium, a
protective lining that covers most of the body's internal organs. Its
most common site is the pleura (outer lining of the lungs and internal
chest wall), but it may also occur in the peritoneum (the lining of
the abdominal cavity), the heart, the pericardium (a sac that
surrounds the heart) or tunica vaginalis.

Most people who develop Mesothelioma have worked on
jobs where they inhaled asbestos
particles, or they have been exposed
to asbestos
dust and fiber in other ways. Washing the clothes of a
family member who worked with asbestos
can also put a person at risk
for developing mesothelioma. Unlike lung cancer, there is no
association between Mesothelioma and smoking.
Compensation via asbestos
funds or lawsuits is an important issue in
mesothelioma (see asbestos
and the law).

The symptoms of Mesothelioma include
shortness of breath due to pleural effusion (fluid between the lung
and the chest wall) or chest wall pain, and general symptoms such as
weight loss. The diagnosis may be suspected with chest X-ray and CT
scan, and is confirmed with a biopsy (tissue sample) and microscopic
examination. A thoracoscopy (inserting a tube with a camera into the
chest) can be used to take biopsies. It allows the introduction of
substances such as talc to obliterate the pleural space (called
pleurodesis), which prevents more fluid from accumulating and pressing
on the lung. Despite treatment with chemotherapy, radiation therapy or
sometimes surgery, the disease carries a poor prognosis. Research
about screening tests for the early detection of mesothelioma is
ongoing.
Symptoms of Mesothelioma
may not appear until 20 to 50 years after exposure to asbestos.
Shortness of breath, cough, and pain in the chest due to an
accumulation of fluid in the pleural space are often symptoms of
pleuralMesothelioma

Symptoms of peritoneal Mesothelioma include weight
loss and cachexia, abdominal swelling and pain due to ascites (a
buildup of fluid in the abdominal cavity). Other symptoms of
peritoneal Mesothelioma
may include bowel obstruction, blood clotting abnormalities, anemia,
and fever. If the cancer has spread beyond the mesothelium to other
parts of the body, symptoms may include pain, trouble swallowing, or
swelling of the neck or face.

These symptoms may be caused by Mesothelioma or by other,
less serious conditions.

Mesothelioma
that affects
the pleura can cause these signs and symptoms:

chest wall pain
pleural effusion, or fluid surrounding the lung
shortness of breath
fatigue or anemia
wheezing, hoarseness, or cough
blood in the sputum (fluid) coughed up (hemoptysis)
In severe cases, the person may have many tumor masses. The individual
may develop a pneumothorax, or collapse of the lung. The disease may
metastasize, or spread, to other parts of the body.

Tumors that affect the abdominal cavity often do not cause symptoms
until they are at a late stage. Symptoms include:

abdominal pain
ascites, or an abnormal buildup of fluid in the abdomen
a mass in the abdomen
problems with bowel function
weight loss
In severe cases of the disease, the following signs and symptoms may be present:

blood clots in the veins, which may cause thrombophlebitis
disseminated intravascular coagulation, a disorder causing severe
bleeding in many body organs
jaundice, or yellowing of the eyes and skin
low blood sugar level
pleural effusion
pulmonary emboli, or blood clots in the arteries of the lungs
severe ascites
A Mesothelioma
does not
usually spread to the bone, brain, or adrenal glands. Pleural tumors
are usually found only on one side of the lungs.
Diagnosing Mesotheliomais
often difficult, because the symptoms are similar to those of a
number of other conditions. Diagnosis begins with a review of the
patient's medical history. A history of exposure to asbestos may
increase clinical suspicion for mesothelioma. A physical examination
is performed, followed by chest X-ray and often lung function tests.
The X-ray may reveal pleural thickening commonly seen after asbestos

exposure and increases suspicion of Mesothelioma A CT (or CAT)
scan or an MRI is usually performed. If a large amount of fluid is
present, abnormal cells may be detected by cytology if this fluid is
aspirated with a syringe. For pleural fluid this is done by a pleural
tap or chest drain, in ascites with an paracentesis or ascitic drain
and in a pericardial effusion with pericardiocentesis. While absence
of malignant cells on cytology does not completely exclude
mesothelioma, it makes it much more unlikely, especially if an
alternative diagnosis can be made (e.g. tuberculosis, heart failure).

If cytology is positive or a plaque is regarded as suspicious, a
biopsy is needed to confirm a diagnosis of Mesothelioma A doctor
removes a sample of tissue for examination under a microscope by a
pathologist. A biopsy may be done in different ways, depending on
where the abnormal area is located. If the cancer is in the chest, the
doctor may perform a thoracoscopy. In this procedure, the doctor makes
a small cut through the chest wall and puts a thin, lighted tube
called a thoracoscope into the chest between two ribs. Thoracoscopy
allows the doctor to look inside the chest and obtain tissue samples.

If the cancer is in the abdomen, the doctor may perform a laparoscopy.
To obtain tissue for examination, the doctor makes a small incision in
the abdomen and inserts a special instrument into the abdominal
cavity. If these procedures do not yield enough tissue, more extensive
diagnostic surgery may be necessary
The mesothelium consists of a single layer of flattened to cuboidal
cells forming the epithelial lining of the serous cavities of the body
including the peritoneal, pericardial and pleural cavities. Deposition
of asbestos fibres in the parenchyma of the lung may result in the
penetration of the visceral pleura from where the fibre can then be
carried to the pleural surface, thus leading to the development of
malignant mesothelial plaques. The processes leading to the
development of peritoneal Mesothelioma remain
unresolved, although it has been proposed that asbestos
fibres from
the lung are transported to the abdomen and associated organs via the
lymphatic system. Additionally, asbestos
fibres may be deposited in
the gut after ingestion of sputum contaminated with asbestos
fibres.

Pleural contamination with asbestos
or other mineral fibres has been
shown to cause cancer. Long thin asbestos
fibers (blue asbestos
,
amphibole fibers) are more potent carcinogens than "feathery fibers"
(chrysotile or white asbestos
fibers).[6] However, there is now
evidence that smaller particles may be more dangerous than the larger
fibers. They remain suspended in the air where they can be inhaled,
and may penetrate more easily and deeper into the lungs. "We probably
will find out a lot more about the health aspects of asbestos
from
[the World Trade Center attack], unfortunately," said Dr. Alan Fein,
chief of pulmonary and critical-care medicine at North Shore-Long
Island Jewish Health System. Dr. Fein has treated several patients for
"World Trade Center syndrome" or respiratory ailments from brief
exposures of only a day or two near the collapsed buildings.

Mesothelioma
development
in rats has been demonstrated following intra-pleural inoculation of
phosphorylated chrysotile fibres. It has been suggested that in
humans, transport of fibres to the pleura is critical to the
pathogenesis of mesothelioma. This is supported by the observed
recruitment of significant numbers of macrophages and other cells of
the immune system to localised lesions of accumulated asbestos fibres
in the pleural and peritoneal cavities of rats. These lesions
continued to attract and accumulate macrophages as the disease
progressed, and cellular changes within the lesion culminated in a
morphologically malignant tumour.

Experimental evidence suggests that asbestos
acts as a complete
carcinogen with the development of Mesothelioma occurring in
sequential stages of initiation and promotion. The molecular
mechanisms underlying the malignant transformation of normal
mesothelial cells by asbestos
fibres remain unclear despite the
demonstration of its oncogenic capabilities. However, complete in
vitro transformation of normal human mesothelial cells to malignant
phenotype following exposure to asbestos
fibres has not yet been
achieved. In general, asbestos
fibres are thought to act through
direct physical interactions with the cells of the mesothelium in
conjunction with indirect effects following interaction with
inflammatory cells such as macrophages.

Analysis of the interactions between asbestos
fibres and DNA has shown
that phagocytosed fibres are able to make contact with chromosomes,
often adhering to the chromatin fibres or becoming entangled within
the chromosome. This contact between the asbestos
fibre and the
chromosomes or structural proteins of the spindle apparatus can induce
complex abnormalities. The most common abnormality is monosomy of
chromosome 22. Other frequent abnormalities include structural
rearrangement of 1p, 3p, 9p and 6q chromosome arms.
asbestos
has also been shown to mediate the entry of foreign DNA into
target cells. Incorporation of this foreign DNA may lead to mutations
and oncogenesis by several possible mechanisms:

Inactivation of tumor suppressor genes
Activation of oncogenes
Activation of proto-oncogenes due to incorporation of foreign DNA
containing a promoter region
Activation of DNA repair enzymes, which may be prone to error
Activation of telomerase
Prevention of apoptosis
asbestos
fibers have been shown to alter the function and secretory
properties of macrophages, ultimately creating conditions which favour
the development of mesothelioma. Following asbestos
phagocytosis,
macrophages generate increased amounts of hydroxyl radicals, which are
normal by-products of cellular anaerobic metabolism. However, these
free radicals are also known clastogenic and membrane-active agents
thought to promote asbestos
carcinogenicity. These oxidants can
participate in the oncogenic process by directly and indirectly
interacting with DNA, modifying membrane-associated cellular events,
including oncogene activation and perturbation of cellular antioxidant
defences.

asbestos
also may possess immunosuppressive properties. For example,
chrysotile fibres have been shown to depress the in vitro
proliferation of phytohemagglutinin-stimulated peripheral blood
lymphocytes, suppress natural killer cell lysis and significantly
reduce lymphokine-activated killer cell viability and recovery.
Furthermore, genetic alterations in asbestos
-activated macrophages may
result in the release of potent mesothelial cell mitogens such as
platelet-derived growth factor (PDGF) and transforming growth factor
which in turn, may induce the chronic stimulation and proliferation of
mesothelial cells after injury by asbestos
fibres

Incidence
Although reported incidence rates have increased in the past 20 years,
mesothelioma is still a relatively rare cancer. The incidence rate is
approximately one per 1,000,000. The highest incidence is found in
Britain, Australia and Belgium: 30 per 1,000,000 per year.[7] For
comparison, populations with high levels of smoking can have a lung
cancer incidence of over 1,000 per 1,000,000. Incidence of malignant
asbestos
currently
ranges from about 7 to 40 per 1,000,000 in industrialized Western
nations, depending on the amount of asbestos
exposure of the
populations during the past several decades.[8] It has been estimated
that incidence may have peaked at 15 per 1,000,000 in the United
States in 2004. Incidence is expected to continue increasing in other
parts of the world. Mesothelioma occurs more
often in men than in women and risk increases with age, but this
disease can appear in either men or women at any age. Approximately
one fifth to one third of all mesotheliomas are peritoneal.

Between 1940 and 1979, approximately 27.5 million people were
occupationally exposed to asbestos in the United States [4]. Between
1973 and 1984, there has been a threefold increase in the diagnosis of
pleural Mesothelioma
in
Caucasian males. From 1980 to the late 1990s, the death rate from Mesothelioma in the USA
increased from 2,000 per year to 3,000, with men four times more
likely to acquire it than women. These rates may not be accurate,
since it is possible that many cases of Mesothelioma are
misdiagnosed as adenocarcinoma of the lung, which is difficult to
differentiate from mesothelioma.

Risk factors
Working with asbestos is the major risk factor for Mesothelioma A history of
asbestos exposure exists in almost all cases. However, Mesothelioma has been
reported in some individuals without any known exposure to asbestos

In rare cases, Mesothelioma has also been
associated with irradiation, intrapleural thorium dioxide
(Thorotrast), and inhalation of other fibrous silicates, such as
erionite.

Asbestos is the name of a group of minerals that occur naturally as
masses of strong, flexible fibers that can be separated into thin
threads and woven. Asbestos has been widely used in many industrial
products, including cement, brake linings, roof shingles, flooring
products, textiles, and insulation. If tiny asbestos particles float
in the air, especially during the manufacturing process, they may be
inhaled or swallowed, and can cause serious health problems. In
addition to Mesothelioma
exposure to asbestos
increases the risk of lung cancer, asbestosis (a
noncancerous, chronic lung ailment), and other cancers, such as those
of the larynx and kidney.

The combination of smoking and asbestos
exposure significantly
increases a person's risk of developing cancer of the airways (lung
cancer, bronchial carcinoma). The Kent brand of cigarettes used
asbestos in its filters for the first few years of production in the
1950s and some cases of Mesothelioma have resulted.
Smoking modern cigarettes does not appear to increase the risk of
mesothelioma.

Some studies suggest that simian virus 40 (SV40) may act as a cofactor
in the development of Mesothelioma
Exposure
asbestos
was known in antiquity, but it wasn't mined and widely used
commercially until the late 1800s. Its use greatly increased during
World War II. Since the early 1940s, millions of American workers have
been exposed to asbestos dust. Initially, the risks associated with
asbestos exposure were not publicly known. However, an increased risk
of developing Mesothelioma was later found
among shipyard workers, people who work in asbestos
mines and mills,
producers of asbestos
products, workers in the heating and
construction industries, and other tradespeople. Today, the U.S.
Occupational Safety and Health Administration (OSHA) sets limits for
acceptable levels of asbestos
exposure in the workplace, and created
guidelines for engineering controls and respirators, protective
clothing, exposure monitoring, hygiene facilities and practices,
warning signs, labeling, recordkeeping, and medical exams. By
contrast, the British Government's Health and Safety Executive (HSE)
states formally that any threshold for Mesothelioma must be at a
very low level and it is widely agreed that if any such threshold does
exist at all, then it cannot currently be quantified. For practical
purposes, therefore, HSE does not assume that any such threshold
exists. People who work with asbestos
wear personal protective
equipment to lower their risk of exposure. Recent findings have shown
that a mineral called erionite has been known to cause genetically
pre-dispositioned individuals to have malignant Mesothelioma rates much
higher than those not pre-dispositioned genetically. A study in
Cappadocia, Turkey has shown that 3 villiages in Turkey have death
rates of 51% attributed to erionite related mesothelioma.

Occupational
Exposure to asbestos
fibres has been recognised as an occupational
health hazard since the early 1900s. Several epidemiological studies
have associated exposure to asbestos with the development of lesions
such as asbestos
bodies in the sputum, pleural plaques, diffuse
pleural thickening, asbestosis, carcinoma of the lung and larynx,
gastrointestinal tumours, and diffuse Mesothelioma of the pleura
and peritoneum.

The documented presence of asbestos
fibres in water supplies and food
products has fostered concerns about the possible impact of long-term
and, as yet, unknown exposure of the general population to these
fibres. Although many authorities consider brief or transient exposure
to asbestos fibres as inconsequential and an unlikely risk factor,
some epidemiologists claim that there is no risk threshold. Cases of
Mesothelioma
have been
found in people whose only exposure was breathing the air through
ventilation systems. Other cases had very minimal (3 months or less)
direct exposure.

Commercial asbestos mining at Wittenoom, Western Australia, occurred
between 1945 and 1966. A cohort study of miners employed at the mine
reported that while no deaths occurred within the first 10 years after
crocidolite exposure, 85 deaths attributable to Mesothelioma had occurred by
1985. By 1994, 539 reported deaths due to mesothelioma had been
reported in Western Australia.

Paraoccupational secondary exposure
Family members and others living with asbestos
workers have an
increased risk of developing Mesothelioma and possibly
other asbestos related diseases. This risk may be the result of
exposure to asbestos
dust brought home on the clothing and hair of
asbestos workers. To reduce the chance of exposing family members to
asbestos fibres, asbestos
workers are usually required to shower and
change their clothing before leaving the workplace.

Asbestos in buildings
Many building materials used in both public and domestic premises
prior to the banning of asbestos may contain asbestos
. Those
performing renovation works or DIY activities may expose themselves to
asbestos dust. In the UK use of Chrysotile asbestos
was banned at the
end of 1999. Brown and blue asbestos
was banned in the UK around 1985.
Buildings built or renovated prior to these dates may contain asbestos

materials.
Environmental exposures
Incidence of mesothelioma had been found to be higher in populations
living near naturally occurring asbestos
. For example, in Cappadocia,
Turkey, an unprecedented Mesothelioma epidemic caused
50% of all deaths in three small villages. Initially, this was
attributed to erionite, however, recently, it has been shown that
erionite causes mesothelioma mostly in families with a genetic
predisposition
Treatment of malignant Mesothelioma using
conventional therapies in combination with radiation and or
chemotherapy on stage I or II Mesothelioma have proved on
average 74.6 percent successful in extending the patients life span by
five years or more [commonly known as remission][this percentage may
increases or decrease depending on date of discovery / stage of
malignant development] (Oncology Today, 2009). Treatment course is
primarily determined by the staging or development. This is unlike
traditional treatment such as surgery by itself which has proved only
be 16.3 percent likely to extend a patients life span by five years or
more [commonly known as remission]. Clinical behavior of the
malignancy is affected by several factors including the continuous
mesothelial surface of the pleural cavity which favors local
metastasis via exfoliated cells, invasion to underlying tissue and
other organs within the pleural cavity, and the extremely long latency
period between asbestos
exposure and development of the disease

5:16 PM  
Anonymous Anonymous said...

asbestos


Mesothelioma
is a form of
cancer that is almost always caused by previous exposure to asbestos.
In this disease, malignant cells develop in the mesothelium, a
protective lining that covers most of the body's internal organs. Its
most common site is the pleura (outer lining of the lungs and internal
chest wall), but it may also occur in the peritoneum (the lining of
the abdominal cavity), the heart, the pericardium (a sac that
surrounds the heart) or tunica vaginalis.

Most people who develop Mesothelioma have worked on
jobs where they inhaled asbestos
particles, or they have been exposed
to asbestos
dust and fiber in other ways. Washing the clothes of a
family member who worked with asbestos
can also put a person at risk
for developing mesothelioma. Unlike lung cancer, there is no
association between Mesothelioma and smoking.
Compensation via asbestos
funds or lawsuits is an important issue in
mesothelioma (see asbestos
and the law).

The symptoms of Mesothelioma include
shortness of breath due to pleural effusion (fluid between the lung
and the chest wall) or chest wall pain, and general symptoms such as
weight loss. The diagnosis may be suspected with chest X-ray and CT
scan, and is confirmed with a biopsy (tissue sample) and microscopic
examination. A thoracoscopy (inserting a tube with a camera into the
chest) can be used to take biopsies. It allows the introduction of
substances such as talc to obliterate the pleural space (called
pleurodesis), which prevents more fluid from accumulating and pressing
on the lung. Despite treatment with chemotherapy, radiation therapy or
sometimes surgery, the disease carries a poor prognosis. Research
about screening tests for the early detection of mesothelioma is
ongoing.
Symptoms of Mesothelioma
may not appear until 20 to 50 years after exposure to asbestos.
Shortness of breath, cough, and pain in the chest due to an
accumulation of fluid in the pleural space are often symptoms of
pleuralMesothelioma

Symptoms of peritoneal Mesothelioma include weight
loss and cachexia, abdominal swelling and pain due to ascites (a
buildup of fluid in the abdominal cavity). Other symptoms of
peritoneal Mesothelioma
may include bowel obstruction, blood clotting abnormalities, anemia,
and fever. If the cancer has spread beyond the mesothelium to other
parts of the body, symptoms may include pain, trouble swallowing, or
swelling of the neck or face.

These symptoms may be caused by Mesothelioma or by other,
less serious conditions.

Mesothelioma
that affects
the pleura can cause these signs and symptoms:

chest wall pain
pleural effusion, or fluid surrounding the lung
shortness of breath
fatigue or anemia
wheezing, hoarseness, or cough
blood in the sputum (fluid) coughed up (hemoptysis)
In severe cases, the person may have many tumor masses. The individual
may develop a pneumothorax, or collapse of the lung. The disease may
metastasize, or spread, to other parts of the body.

Tumors that affect the abdominal cavity often do not cause symptoms
until they are at a late stage. Symptoms include:

abdominal pain
ascites, or an abnormal buildup of fluid in the abdomen
a mass in the abdomen
problems with bowel function
weight loss
In severe cases of the disease, the following signs and symptoms may be present:

blood clots in the veins, which may cause thrombophlebitis
disseminated intravascular coagulation, a disorder causing severe
bleeding in many body organs
jaundice, or yellowing of the eyes and skin
low blood sugar level
pleural effusion
pulmonary emboli, or blood clots in the arteries of the lungs
severe ascites
A Mesothelioma
does not
usually spread to the bone, brain, or adrenal glands. Pleural tumors
are usually found only on one side of the lungs.
Diagnosing Mesotheliomais
often difficult, because the symptoms are similar to those of a
number of other conditions. Diagnosis begins with a review of the
patient's medical history. A history of exposure to asbestos may
increase clinical suspicion for mesothelioma. A physical examination
is performed, followed by chest X-ray and often lung function tests.
The X-ray may reveal pleural thickening commonly seen after asbestos

exposure and increases suspicion of Mesothelioma A CT (or CAT)
scan or an MRI is usually performed. If a large amount of fluid is
present, abnormal cells may be detected by cytology if this fluid is
aspirated with a syringe. For pleural fluid this is done by a pleural
tap or chest drain, in ascites with an paracentesis or ascitic drain
and in a pericardial effusion with pericardiocentesis. While absence
of malignant cells on cytology does not completely exclude
mesothelioma, it makes it much more unlikely, especially if an
alternative diagnosis can be made (e.g. tuberculosis, heart failure).

If cytology is positive or a plaque is regarded as suspicious, a
biopsy is needed to confirm a diagnosis of Mesothelioma A doctor
removes a sample of tissue for examination under a microscope by a
pathologist. A biopsy may be done in different ways, depending on
where the abnormal area is located. If the cancer is in the chest, the
doctor may perform a thoracoscopy. In this procedure, the doctor makes
a small cut through the chest wall and puts a thin, lighted tube
called a thoracoscope into the chest between two ribs. Thoracoscopy
allows the doctor to look inside the chest and obtain tissue samples.

If the cancer is in the abdomen, the doctor may perform a laparoscopy.
To obtain tissue for examination, the doctor makes a small incision in
the abdomen and inserts a special instrument into the abdominal
cavity. If these procedures do not yield enough tissue, more extensive
diagnostic surgery may be necessary
The mesothelium consists of a single layer of flattened to cuboidal
cells forming the epithelial lining of the serous cavities of the body
including the peritoneal, pericardial and pleural cavities. Deposition
of asbestos fibres in the parenchyma of the lung may result in the
penetration of the visceral pleura from where the fibre can then be
carried to the pleural surface, thus leading to the development of
malignant mesothelial plaques. The processes leading to the
development of peritoneal Mesothelioma remain
unresolved, although it has been proposed that asbestos
fibres from
the lung are transported to the abdomen and associated organs via the
lymphatic system. Additionally, asbestos
fibres may be deposited in
the gut after ingestion of sputum contaminated with asbestos
fibres.

Pleural contamination with asbestos
or other mineral fibres has been
shown to cause cancer. Long thin asbestos
fibers (blue asbestos
,
amphibole fibers) are more potent carcinogens than "feathery fibers"
(chrysotile or white asbestos
fibers).[6] However, there is now
evidence that smaller particles may be more dangerous than the larger
fibers. They remain suspended in the air where they can be inhaled,
and may penetrate more easily and deeper into the lungs. "We probably
will find out a lot more about the health aspects of asbestos
from
[the World Trade Center attack], unfortunately," said Dr. Alan Fein,
chief of pulmonary and critical-care medicine at North Shore-Long
Island Jewish Health System. Dr. Fein has treated several patients for
"World Trade Center syndrome" or respiratory ailments from brief
exposures of only a day or two near the collapsed buildings.

Mesothelioma
development
in rats has been demonstrated following intra-pleural inoculation of
phosphorylated chrysotile fibres. It has been suggested that in
humans, transport of fibres to the pleura is critical to the
pathogenesis of mesothelioma. This is supported by the observed
recruitment of significant numbers of macrophages and other cells of
the immune system to localised lesions of accumulated asbestos fibres
in the pleural and peritoneal cavities of rats. These lesions
continued to attract and accumulate macrophages as the disease
progressed, and cellular changes within the lesion culminated in a
morphologically malignant tumour.

Experimental evidence suggests that asbestos
acts as a complete
carcinogen with the development of Mesothelioma occurring in
sequential stages of initiation and promotion. The molecular
mechanisms underlying the malignant transformation of normal
mesothelial cells by asbestos
fibres remain unclear despite the
demonstration of its oncogenic capabilities. However, complete in
vitro transformation of normal human mesothelial cells to malignant
phenotype following exposure to asbestos
fibres has not yet been
achieved. In general, asbestos
fibres are thought to act through
direct physical interactions with the cells of the mesothelium in
conjunction with indirect effects following interaction with
inflammatory cells such as macrophages.

Analysis of the interactions between asbestos
fibres and DNA has shown
that phagocytosed fibres are able to make contact with chromosomes,
often adhering to the chromatin fibres or becoming entangled within
the chromosome. This contact between the asbestos
fibre and the
chromosomes or structural proteins of the spindle apparatus can induce
complex abnormalities. The most common abnormality is monosomy of
chromosome 22. Other frequent abnormalities include structural
rearrangement of 1p, 3p, 9p and 6q chromosome arms.
asbestos
has also been shown to mediate the entry of foreign DNA into
target cells. Incorporation of this foreign DNA may lead to mutations
and oncogenesis by several possible mechanisms:

Inactivation of tumor suppressor genes
Activation of oncogenes
Activation of proto-oncogenes due to incorporation of foreign DNA
containing a promoter region
Activation of DNA repair enzymes, which may be prone to error
Activation of telomerase
Prevention of apoptosis
asbestos
fibers have been shown to alter the function and secretory
properties of macrophages, ultimately creating conditions which favour
the development of mesothelioma. Following asbestos
phagocytosis,
macrophages generate increased amounts of hydroxyl radicals, which are
normal by-products of cellular anaerobic metabolism. However, these
free radicals are also known clastogenic and membrane-active agents
thought to promote asbestos
carcinogenicity. These oxidants can
participate in the oncogenic process by directly and indirectly
interacting with DNA, modifying membrane-associated cellular events,
including oncogene activation and perturbation of cellular antioxidant
defences.

asbestos
also may possess immunosuppressive properties. For example,
chrysotile fibres have been shown to depress the in vitro
proliferation of phytohemagglutinin-stimulated peripheral blood
lymphocytes, suppress natural killer cell lysis and significantly
reduce lymphokine-activated killer cell viability and recovery.
Furthermore, genetic alterations in asbestos
-activated macrophages may
result in the release of potent mesothelial cell mitogens such as
platelet-derived growth factor (PDGF) and transforming growth factor
which in turn, may induce the chronic stimulation and proliferation of
mesothelial cells after injury by asbestos
fibres

Incidence
Although reported incidence rates have increased in the past 20 years,
mesothelioma is still a relatively rare cancer. The incidence rate is
approximately one per 1,000,000. The highest incidence is found in
Britain, Australia and Belgium: 30 per 1,000,000 per year.[7] For
comparison, populations with high levels of smoking can have a lung
cancer incidence of over 1,000 per 1,000,000. Incidence of malignant
asbestos
currently
ranges from about 7 to 40 per 1,000,000 in industrialized Western
nations, depending on the amount of asbestos
exposure of the
populations during the past several decades.[8] It has been estimated
that incidence may have peaked at 15 per 1,000,000 in the United
States in 2004. Incidence is expected to continue increasing in other
parts of the world. Mesothelioma occurs more
often in men than in women and risk increases with age, but this
disease can appear in either men or women at any age. Approximately
one fifth to one third of all mesotheliomas are peritoneal.

Between 1940 and 1979, approximately 27.5 million people were
occupationally exposed to asbestos in the United States [4]. Between
1973 and 1984, there has been a threefold increase in the diagnosis of
pleural Mesothelioma
in
Caucasian males. From 1980 to the late 1990s, the death rate from Mesothelioma in the USA
increased from 2,000 per year to 3,000, with men four times more
likely to acquire it than women. These rates may not be accurate,
since it is possible that many cases of Mesothelioma are
misdiagnosed as adenocarcinoma of the lung, which is difficult to
differentiate from mesothelioma.

Risk factors
Working with asbestos is the major risk factor for Mesothelioma A history of
asbestos exposure exists in almost all cases. However, Mesothelioma has been
reported in some individuals without any known exposure to asbestos

In rare cases, Mesothelioma has also been
associated with irradiation, intrapleural thorium dioxide
(Thorotrast), and inhalation of other fibrous silicates, such as
erionite.

Asbestos is the name of a group of minerals that occur naturally as
masses of strong, flexible fibers that can be separated into thin
threads and woven. Asbestos has been widely used in many industrial
products, including cement, brake linings, roof shingles, flooring
products, textiles, and insulation. If tiny asbestos particles float
in the air, especially during the manufacturing process, they may be
inhaled or swallowed, and can cause serious health problems. In
addition to Mesothelioma
exposure to asbestos
increases the risk of lung cancer, asbestosis (a
noncancerous, chronic lung ailment), and other cancers, such as those
of the larynx and kidney.

The combination of smoking and asbestos
exposure significantly
increases a person's risk of developing cancer of the airways (lung
cancer, bronchial carcinoma). The Kent brand of cigarettes used
asbestos in its filters for the first few years of production in the
1950s and some cases of Mesothelioma have resulted.
Smoking modern cigarettes does not appear to increase the risk of
mesothelioma.

Some studies suggest that simian virus 40 (SV40) may act as a cofactor
in the development of Mesothelioma
Exposure
asbestos
was known in antiquity, but it wasn't mined and widely used
commercially until the late 1800s. Its use greatly increased during
World War II. Since the early 1940s, millions of American workers have
been exposed to asbestos dust. Initially, the risks associated with
asbestos exposure were not publicly known. However, an increased risk
of developing Mesothelioma was later found
among shipyard workers, people who work in asbestos
mines and mills,
producers of asbestos
products, workers in the heating and
construction industries, and other tradespeople. Today, the U.S.
Occupational Safety and Health Administration (OSHA) sets limits for
acceptable levels of asbestos
exposure in the workplace, and created
guidelines for engineering controls and respirators, protective
clothing, exposure monitoring, hygiene facilities and practices,
warning signs, labeling, recordkeeping, and medical exams. By
contrast, the British Government's Health and Safety Executive (HSE)
states formally that any threshold for Mesothelioma must be at a
very low level and it is widely agreed that if any such threshold does
exist at all, then it cannot currently be quantified. For practical
purposes, therefore, HSE does not assume that any such threshold
exists. People who work with asbestos
wear personal protective
equipment to lower their risk of exposure. Recent findings have shown
that a mineral called erionite has been known to cause genetically
pre-dispositioned individuals to have malignant Mesothelioma rates much
higher than those not pre-dispositioned genetically. A study in
Cappadocia, Turkey has shown that 3 villiages in Turkey have death
rates of 51% attributed to erionite related mesothelioma.

Occupational
Exposure to asbestos
fibres has been recognised as an occupational
health hazard since the early 1900s. Several epidemiological studies
have associated exposure to asbestos with the development of lesions
such as asbestos
bodies in the sputum, pleural plaques, diffuse
pleural thickening, asbestosis, carcinoma of the lung and larynx,
gastrointestinal tumours, and diffuse Mesothelioma of the pleura
and peritoneum.

The documented presence of asbestos
fibres in water supplies and food
products has fostered concerns about the possible impact of long-term
and, as yet, unknown exposure of the general population to these
fibres. Although many authorities consider brief or transient exposure
to asbestos fibres as inconsequential and an unlikely risk factor,
some epidemiologists claim that there is no risk threshold. Cases of
Mesothelioma
have been
found in people whose only exposure was breathing the air through
ventilation systems. Other cases had very minimal (3 months or less)
direct exposure.

Commercial asbestos mining at Wittenoom, Western Australia, occurred
between 1945 and 1966. A cohort study of miners employed at the mine
reported that while no deaths occurred within the first 10 years after
crocidolite exposure, 85 deaths attributable to Mesothelioma had occurred by
1985. By 1994, 539 reported deaths due to mesothelioma had been
reported in Western Australia.

Paraoccupational secondary exposure
Family members and others living with asbestos
workers have an
increased risk of developing Mesothelioma and possibly
other asbestos related diseases. This risk may be the result of
exposure to asbestos
dust brought home on the clothing and hair of
asbestos workers. To reduce the chance of exposing family members to
asbestos fibres, asbestos
workers are usually required to shower and
change their clothing before leaving the workplace.

Asbestos in buildings
Many building materials used in both public and domestic premises
prior to the banning of asbestos may contain asbestos
. Those
performing renovation works or DIY activities may expose themselves to
asbestos dust. In the UK use of Chrysotile asbestos
was banned at the
end of 1999. Brown and blue asbestos
was banned in the UK around 1985.
Buildings built or renovated prior to these dates may contain asbestos

materials.
Environmental exposures
Incidence of mesothelioma had been found to be higher in populations
living near naturally occurring asbestos
. For example, in Cappadocia,
Turkey, an unprecedented Mesothelioma epidemic caused
50% of all deaths in three small villages. Initially, this was
attributed to erionite, however, recently, it has been shown that
erionite causes mesothelioma mostly in families with a genetic
predisposition
Treatment of malignant Mesothelioma using
conventional therapies in combination with radiation and or
chemotherapy on stage I or II Mesothelioma have proved on
average 74.6 percent successful in extending the patients life span by
five years or more [commonly known as remission][this percentage may
increases or decrease depending on date of discovery / stage of
malignant development] (Oncology Today, 2009). Treatment course is
primarily determined by the staging or development. This is unlike
traditional treatment such as surgery by itself which has proved only
be 16.3 percent likely to extend a patients life span by five years or
more [commonly known as remission]. Clinical behavior of the
malignancy is affected by several factors including the continuous
mesothelial surface of the pleural cavity which favors local
metastasis via exfoliated cells, invasion to underlying tissue and
other organs within the pleural cavity, and the extremely long latency
period between asbestos
exposure and development of the disease

5:17 PM  

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